#
 
contact

The Ultimate Mold, Mycotoxin and Human Symptom Chart

Not all mold produces secondary metabolites but most of the ones that do are harmful to humans.  These molds can produce these secondary metabolites that are either antibiotics or mycotoxins. Antibiotics are isolated from mold (and some bacterial) cultures and some of their bacteriotoxic or bacteriostatic properties are exploited medicinally to try to fight infections.

Mycotoxins are all cytotoxic, which disrupt various cellular structures such as membranes, and interferes with vital cellular processes such as protein, RNA and DNA synthesis. They are also toxic to the cells of higher plants and animals, including humans.  This chart specifically deals with the effects on human health.

Mold Name Mycotoxin Produced by that mold Route of Exposure Health Effects of that Mycotoxin in the Human Body Possible documented remedies, therapies or treatments
Acremonium crotocinigenum Crotocin Oral, dermal, inhalation, and parenteral (contaminated drugs)
Crotocin is a trichothecene. Trichothecenes are a very large family of chemically related mycotoxins produced by various species of Fusarium, Myrothecium, Trichoderma, Trichothecium, Cephalosporium, Verticimonosporium, and Stachybotrys. Trichothecenes have multiorgan effects including anoerxia and weight loss, growth retardation, nervous disorders, cardiovascular alterations, immunodepression, hemostatic derangements, skin toxicity, decreased reproductive capacity, bone marrow damage, and alimentary toxic aleukia. After direct dermal application or oral ingestion, the trichothecene mycotoxins can cause rapid irritation to the skin or intestinal mucosa, including skin irritation, burning and itching, rash or blisters, and bleeding. Eye contact can cause tearing, eye pain, conjunctivitis, burning sensations about the eyes, and blurred vision for up to 1 week. Symptoms also include nausea, vomiting, fatigue, dyspnea, and acute vascular effects leading to hypotension and shock.
Taking 2 daily warm baths of water and 2 cups of ammonia 3%-household type (clear) over 3 month periods was shown in lab tests to neutralize Trichothecene mycotoxins in humans, infants and animals.
Alternaria alternata Altenuic acid      
Alternaria alternata Alternariol      
Aspergillus carneus Citrinin Oral, dermal, inhalation, and parenteral (contaminated drugs)
Not classifiable as to its carcinogenicity to humans. Citrinin acts as a nephrotoxin. It causes mycotoxic nephropathy and found as the cause of Balkan nephropathy and yellow rice fever in humans. Though the kidney is the major target organ of citrinin toxicity, other target organs such as liver and bone marrow have also been reported. Irritation of the eyes, skin, or respiratory tract, depending on the route of exposure. May produce an allergic hypersensitivity dermatitis or asthma with bronchospasm and wheezing with chronic exposure.
 
Aspergillus carneus Maltoryzine - Maltoryzine may cause peripherall nerve and sensation: Flaccid paralysis without anesthesia (usually neuromuscular blockage)  -
Aspergillus carneus Viomellein - - -
Aspergillus clavatus Cytochalasin E Oral, dermal, inhalation, and parenteral (contaminated drugs).
Can cause cells to undergo apoptosis. Cytochalasin E also inhibits angiogenesis and tumor growth. Major biological effects of cytochalasins include inhibition of the division of cytoplasm, reversible inhibition of cell movement, induction of nuclear extrusion, inhibition of such processes as phagocytosis, platelet aggregation and clot retraction, glucose transport, thyroid secretion, and release of growth hormone. Some cytochalasins have been shown to have developmental effects. Congestive degenerative changes, necrosis of liver, kidney, spleen, pancreas, and small intestine, brain edema, pulmonary hemorrhages, and injury to vascular walls.
 
Aspergillus clavatus Maltoryzine -

Maltoryzine may cause peripherall nerve and sensation: Flaccid paralysis without anesthesia (usually neuromuscular blockage) 

 
Aspergillus clavatus Patulin Oral, dermal, inhalation, and parenteral (contaminated drugs)
gastrointestinal hyperemia, distension, hemorrhage and ulceration. It has also been shown to be immunotoxic (attacks the immune system), and neurotoxic (causing many neurological problems). Skin irritation occurs in cases of topical exposure. Patulin is also a stomach irritant and causes nausea and vomiting if ingested.
 
Aspergillus clavatus Tryptoquivalene - - -
Aspergillus clavatus Viomellein - - -
Aspergillus flavus Aflatrem Oral, dermal, inhalation, and parenteral (contaminated drugs)
Tremorgenic mycotoxins affect central nervous system activity, inducing neurologic symptoms including mental confusion, paralysis, tremors, seizures, and death.
activated charcoal administered can limit further absorption of toxins.

Aspergillus flavus
Aflatoxin Oral, dermal, inhalation, and parenteral (contaminated drugs)
vomiting, abdominal pain, pulmonary edema, convulsions, coma, and death with cerebral edema and fatty involvment of the liver, kidneys, and heart. Liver cell cancers.
 
Aspergillus flavus Maltoryzine -

Maltoryzine may cause peripherall nerve and sensation: Flaccid paralysis without anesthesia (usually neuromuscular blockage) 

 
Aspergillus flavus Sterigmatocystin Oral, dermal, inhalation, and parenteral (contaminated drugs)

Cell damage, carcinogenic to humans, Sterigmatocystin is considered as a potent carcinogen, mutagen and teratogen. It causes necrosis of the liver and kidney. May also cause hepatocellular carcinoma, acute hepatitis, Reye's syndrome, bile duct cell proliferation, periportal fibrosis, hemorrhages, mucous membrane jaundice, fatty liver changes, cirrhosis in malnourished children, and kwashiorkor. anorexia and weight loss. It can also cause diarrhea.

Phenobarbital may be administered to enhances hepatic transformation activities and protect against Sterigmatocystin-induced toxicity, carcinogenicity and DNA binding in vivo. In cases of ingestion, feeding large quantities of an adsorbent such as activated charcoal may be used. The hepatic function should be monitored.
Aspergillus flavus Viomellein - - -
Aspergillus fumigatus

Fumagilin

     
Aspergillus fumigatus Gliotoxin

Oral, dermal, inhalation, and parenteral (contaminated drugs).
Aspergillus fumigatus is case in point. It is the major species associated with aspergillosis and produces gliotoxins (inhibitors of T-cell activation and proliferation as well as macrophage phagocytosis). However, gliotoxin is not known to be produced in significant amounts by Aspergillus fumigatus during human diseas
e (265).

Gliotoxin is a fungal toxin produced by various species of Trichoderma, Gladiocladium fimbriatum, Aspergillus fumigatus, and Penicillium.

Gliotoxin possesses immune system suppression properties as it may cause apoptosis in certain types of cells of the immune system, including neutrophils, eosinophils, granulocytes, macrophages, and thymocytes. It is also cytotoxic and causes neurite degeneration.
gliotoxin has been associated with infections by Candida albicans (230, 231).
The ability to grow at human body temperature (37°C) is clearly an important requirement for systemic mycotic infection, but the optimum temperature for the biosynthesis of most mycotoxins is within a more mesophilic range (20 to 30°C).
 
Aspergillus fumigatus Maltoryzine -

Maltoryzine may cause peripherall nerve and sensation: Flaccid paralysis without anesthesia (usually neuromuscular blockage) 

 
Aspergillus fumigatus Verruculogen Oral, dermal, inhalation, and parenteral (contaminated drugs).
Verruculogen is a tremorgenic mycotoxin that has been found in fungi of the genera Penicillium and Aspergillus. It may be found in contaminated cereal crops such as oats, barley, millet, corn and rice
Tremorgenic mycotoxins affect central nervous system activity.
Verruculogen is also genotoxic and causes DNA damage.
Tremorgenic mycotoxins affect central nervous system activity, inducing neurologic symptoms including mental confusion, paralysis, tremors, seizures, and death. They cause a neurological disease of cattle known as "staggers syndrome", which is characterized by muscle tremors, hyperexcitability, convulsions and ataxia.
To control severe tremors caused by tremorgenic mycotoxins, methocarbamol should be administered. Generalized seizures may be treated with diazepam followed by methocarbamol or a barbiturate such as pentobarbital sodium. Gastric lavage should be performed and activated charcoal administered to limit further absorption of toxins.
Aspergillus fumigatus Viriditoxin - - -
Aspergillus fumigatus Viomellein - - -
Aspergillus nidulans Sterigmatocystin Oral, dermal, inhalation, and parenteral (contaminated drugs) Cell damage, carcinogenic to humans, Sterigmatocystin is considered as a potent carcinogen, mutagen and teratogen. It causes necrosis of the liver and kidney. May also cause hepatocellular carcinoma, acute hepatitis, Reye's syndrome, bile duct cell proliferation, periportal fibrosis, hemorrhages, mucous membrane jaundice, fatty liver changes, cirrhosis in malnourished children, and kwashiorkor. anorexia and weight loss. It can also cause diarrhea. Phenobarbital may be administered to enhances hepatic transformation activities and protect against Sterigmatocystin-induced toxicity, carcinogenicity and DNA binding in vivo. In cases of ingestion, feeding large quantities of an adsorbent such as activated charcoal may be used. The hepatic function should be monitored.
Aspergillus nidulans Maltoryzine -

Maltoryzine may cause peripherall nerve and sensation: Flaccid paralysis without anesthesia (usually neuromuscular blockage) 

 
Aspergillus nidulans Viomellein - - -
Aspergillus niger Malformin - - -
Aspergillus niger Maltoryzine -

Maltoryzine may cause peripherall nerve and sensation: Flaccid paralysis without anesthesia (usually neuromuscular blockage) 

 
Aspergillus niger Oxalic acid Oxalic acid and oxalates are abundantly present in many plants, most notably fat hen (lamb's quarters), sorrel, and Oxalis species. The root and/or leaves of rhubarb and buckwheat are listed being high in oxalic acid.[8] Other edible plants that contain significant concentrations of oxalic acid include—in decreasing order—star fruit (carambola), black pepper, parsley, poppy seed, amaranth, spinach, chard, beets, cocoa, chocolate, most nuts, most berries, and beans. Because it binds vital nutrients such as calcium, long-term consumption of foods high in oxalic acid can be problematic. Healthy individuals can safely consume such foods in moderation, but those with kidney disorders, gout, rheumatoid arthritis, or certain forms of chronic vulvar pain (vulvodynia) are typically advised to avoid foods high in oxalic acid or oxalates. The calcium oxalate precipitate (better known as kidney stones) obstruct the kidney tubules. Conversely, calcium supplements taken along with foods high in oxalic acid can cause calcium oxalate to precipitate out in the gut and drastically reduce the levels of oxalate absorbed by the body (by 97% in some cases.) Chronically high levels of oxalic acid are associated with at least 2 inborn errors of metabolism including: Type I primary hyperoxaluria and Primary hyperoxaluria. Oxalate stones in primary hyperoxaluria tend to be severe, resulting in relatively early kidney damage (before age 20), which impairs the excretion of oxalate leading to a further acceleration in accumulation of oxalate in the body. After the development of renal failure patients may develop oxalate deposits in the bones, joints and bone marrow. Severe cases may develop haematological problems such as anaemia and thrombocytopaenia. The deposition of oxalate in the body is sometimes called "oxalosis" to be distinguished from "oxaluria" which refers to oxalate in the urine.Oxalic acid poisoning symptoms include weakness, burning in the mouth, death from cardiovascular collapse, on the respiratory system, throat – burning in the throat, abdominal pain, nausea, vomiting, diarrhea, convulsions, and coma. Acute Exposure: If oxalic acid is swallowed, immediately give the person water or milk, unless instructed otherwise by a health care provider. DO NOT give water or milk if the person is having symptoms (such as vomiting, convulsions, or a decreased level of alertness) that make it hard to swallow. If acute exposure occurs to the eyes, irrigate opened eyes for several minutes under running water. Chronic exposure: in some patients with primary hyperoxaluria type 1, pyridoxine treatment (vitamin B6) may decrease oxalate excretion and prevent kidney stone formation.
Aspergillus niger Viomellein - - -
Aspergillus ochraceus Destruxin B Found in grains, soil and salted food products. It is not usually associated with decaying vegetation.    
Aspergillus ochraceus Maltoryzine Found in grains, soil and salted food products. It is not usually associated with decaying vegetation. Maltoryzine may cause peripherall nerve and sensation: Flaccid paralysis without anesthesia (usually neuromuscular blockage)   
Aspergillus ochraceus Ochratoxin Found in grains, soil and salted food products. It is not usually associated with decaying vegetation.    
Aspergillus ochraceus Penicillic acid Found in grains, soil and salted food products. It is not usually associated with decaying vegetation. reported to be kidney and liver toxins  
Aspergillus ochraceus Viomellein Found in grains, soil and salted food products. It is not usually associated with decaying vegetation. reported to be kidney and liver toxins  
Aspergillus parasiticus Aflatoxin  
vomiting, abdominal pain, pulmonary edema, convulsions, coma, and death with cerebral edema and fatty involvment of the liver, kidneys, and heart. Liver cell cancers.
 
Aspergillus parasiticus Maltoryzine   Maltoryzine may cause peripherall nerve and sensation: Flaccid paralysis without anesthesia (usually neuromuscular blockage)   
Aspergillus parasiticus Viomellein      
Aspergillus terreus Citrinin   Not classifiable as to its carcinogenicity to humans. Citrinin acts as a nephrotoxin. It causes mycotoxic nephropathy and found as the cause of Balkan nephropathy and yellow rice fever in humans. Though the kidney is the major target organ of citrinin toxicity, other target organs such as liver and bone marrow have also been reported. Irritation of the eyes, skin, or respiratory tract, depending on the route of exposure. May produce an allergic hypersensitivity dermatitis or asthma with bronchospasm and wheezing with chronic exposure.  
Aspergillus terreus Citreoviridin   reported brain neurotoxicity may be related to the altered enzymatic activities.  
Aspergillus terreus Maltoryzine   Maltoryzine may cause peripherall nerve and sensation: Flaccid paralysis without anesthesia (usually neuromuscular blockage)   
Aspergillus terreus Viomellein      
Aspergillus ustus Austdiol      
Aspergillus ustus Austamide      
Aspergillus ustus Austocystin      
Aspergillus ustus Brevianamide      
Aspergillus ustus Maltoryzine   Maltoryzine may cause peripherall nerve and sensation: Flaccid paralysis without anesthesia (usually neuromuscular blockage)   
Aspergillus ustus Viomellein      
Aspergillus versicolor Cyclopiazonic acid      
Aspergillus versicolor Maltoryzine   Maltoryzine may cause peripherall nerve and sensation: Flaccid paralysis without anesthesia (usually neuromuscular blockage)   
Aspergillus versicolor Sterigmatocystin Oral, dermal, inhalation, and parenteral (contaminated drugs) Cell damage, carcinogenic to humans, Sterigmatocystin is considered as a potent carcinogen, mutagen and teratogen. It causes necrosis of the liver and kidney. May also cause hepatocellular carcinoma, acute hepatitis, Reye's syndrome, bile duct cell proliferation, periportal fibrosis, hemorrhages, mucous membrane jaundice, fatty liver changes, cirrhosis in malnourished children, and kwashiorkor. anorexia and weight loss. It can also cause diarrhea. Phenobarbital may be administered to enhances hepatic transformation activities and protect against Sterigmatocystin-induced toxicity, carcinogenicity and DNA binding in vivo. In cases of ingestion, feeding large quantities of an adsorbent such as activated charcoal may be used. The hepatic function should be monitored.
Aspergillus versicolor Viomellein   reported to be kidney and liver toxins  
Botrytis Patulin Oral, dermal, inhalation, and parenteral (contaminated drugs).
gastrointestinal hyperemia, distension, hemorrhage and ulceration. It has also been shown to be immunotoxic (attacks the immune system), and neurotoxic (causing many neurological problems). Skin irritation occurs in cases of topical exposure. Patulin is also a stomach irritant and causes nausea and vomiting if ingested.
 
Cephalasporium Trichothecene Used in antibiotics such as Cephalexin    
Chaetomium cochliodes Cochliodinol - - -
Chaetomium globosum Chaetoglobosin - - -
Cylindrocarpon Roridin E (Trichothecene class) Oral, dermal, inhalation, and parenteral (contaminated drugs). Trichothecenes have multiorgan effects including anoerxia and weight loss, growth retardation, nervous disorders, cardiovascular alterations, immunodepression, hemostatic derangements, skin toxicity, decreased reproductive capacity, bone marrow damage, and alimentary toxic aleukia. After direct dermal application or oral ingestion, the trichothecene mycotoxins can cause rapid irritation to the skin or intestinal mucosa, including skin irritation, burning and itching, rash or blisters, and bleeding. Eye contact can cause tearing, eye pain, conjunctivitis, burning sensations about the eyes, and blurred vision for up to 1 week. Symptoms also include nausea, vomiting, fatigue, dyspnea, and acute vascular effects leading to hypotension and shock. Taking 2 daily warm baths of water and 2 cups of ammonia 3%-household type (clear) over 3 month periods was shown in lab tests to remove and neutralize Trichothecene mycotoxins in humans, infants and animals.
Dendrodochium Verrucarin A, B or J (Trichothecene class) Oral, dermal, inhalation, and parenteral (contaminated drugs).
Verrucarin A is a trichothecene. Trichothecenes are a very large family of chemically related mycotoxins produced by various species of Fusarium, Myrothecium, Trichoderma, Trichothecium, Cephalosporium, Verticimonosporium, and Stachybotrys. The most important structural features causing the biological activities of trichothecenes are: the 12,13-epoxy ring, the presence of hydroxyl or acetyl groups at appropriate positions on the trichothecene nucleus and the structure and position of the side-chain. They are produced on many different grains like wheat, oats or maize by various Fusarium species such as F. graminearum, F. sporotrichioides, F. poae and F. equiseti. Some molds that produce trichothecene mycotoxins, such as Stachybotrys chartarum, can grow in damp indoor environments and may contribute to health problems among building occupants.
Trichothecenes have multiorgan effects including anoerxia and weight loss, growth retardation, nervous disorders, cardiovascular alterations, immunodepression, hemostatic derangements, skin toxicity, decreased reproductive capacity, bone marrow damage, and alimentary toxic aleukia. After direct dermal application or oral ingestion, the trichothecene mycotoxins can cause rapid irritation to the skin or intestinal mucosa, including skin irritation, burning and itching, rash or blisters, and bleeding. Eye contact can cause tearing, eye pain, conjunctivitis, burning sensations about the eyes, and blurred vision for up to 1 week. Symptoms also include nausea, vomiting, fatigue, dyspnea, and acute vascular effects leading to hypotension and shock. Taking 2 daily warm baths of water and 2 cups of ammonia 3%-household type (clear) over 3 month periods was shown in lab tests to remove and neutralize Trichothecene mycotoxins in humans, infants and animals.
Eurotium chevalieri Xanthocillin      
Fusarium avenaceum Yavanicin+1      
Fusarium avenaceum

Acetoxyscirpenediol
(Trichothecene class)

Oral, dermal, inhalation, and parenteral (contaminated drugs).
Verrucarin A is a trichothecene. Trichothecenes are a very large family of chemically related mycotoxins produced by various species of Fusarium, Myrothecium, Trichoderma, Trichothecium, Cephalosporium, Verticimonosporium, and Stachybotrys. The most important structural features causing the biological activities of trichothecenes are: the 12,13-epoxy ring, the presence of hydroxyl or acetyl groups at appropriate positions on the trichothecene nucleus and the structure and position of the side-chain. They are produced on many different grains like wheat, oats or maize by various Fusarium species such as F. graminearum, F. sporotrichioides, F. poae and F. equiseti. Some molds that produce trichothecene mycotoxins, such as Stachybotrys chartarum, can grow in damp indoor environments and may contribute to health problems among building occupants.
Trichothecenes have multiorgan effects including anoerxia and weight loss, growth retardation, nervous disorders, cardiovascular alterations, immunodepression, hemostatic derangements, skin toxicity, decreased reproductive capacity, bone marrow damage, and alimentary toxic aleukia. After direct dermal application or oral ingestion, the trichothecene mycotoxins can cause rapid irritation to the skin or intestinal mucosa, including skin irritation, burning and itching, rash or blisters, and bleeding. Eye contact can cause tearing, eye pain, conjunctivitis, burning sensations about the eyes, and blurred vision for up to 1 week. Symptoms also include nausea, vomiting, fatigue, dyspnea, and acute vascular effects leading to hypotension and shock. Taking 2 daily warm baths of water and 2 cups of ammonia 3%-household type (clear) over 3 month periods was shown in lab tests to remove and neutralize Trichothecene mycotoxins in humans, infants and animals.
Fusarium avenaceum Acetyldeoxynivalenol Verrucarin A is a trichothecene. Trichothecenes are a very large family of chemically related mycotoxins produced by various species of Fusarium, Myrothecium, Trichoderma, Trichothecium, Cephalosporium, Verticimonosporium, and Stachybotrys. The most important structural features causing the biological activities of trichothecenes are: the 12,13-epoxy ring, the presence of hydroxyl or acetyl groups at appropriate positions on the trichothecene nucleus and the structure and position of the side-chain. They are produced on many different grains like wheat, oats or maize by various Fusarium species such as F. graminearum, F. sporotrichioides, F. poae and F. equiseti. Some molds that produce trichothecene mycotoxins, such as Stachybotrys chartarum, can grow in damp indoor environments and may contribute to health problems among building occupants. Trichothecenes have multiorgan effects including anoerxia and weight loss, growth retardation, nervous disorders, cardiovascular alterations, immunodepression, hemostatic derangements, skin toxicity, decreased reproductive capacity, bone marrow damage, and alimentary toxic aleukia. After direct dermal application or oral ingestion, the trichothecene mycotoxins can cause rapid irritation to the skin or intestinal mucosa, including skin irritation, burning and itching, rash or blisters, and bleeding. Eye contact can cause tearing, eye pain, conjunctivitis, burning sensations about the eyes, and blurred vision for up to 1 week. Symptoms also include nausea, vomiting, fatigue, dyspnea, and acute vascular effects leading to hypotension and shock. Taking 2 daily warm baths of water and 2 cups of ammonia 3%-household type (clear) over 3 month periods was shown in lab tests to remove and neutralize Trichothecene mycotoxins in humans, infants and animals.
Fusarium culmorum Yavanicin+1      

Fusarium culmorum

Acetoxyscirpenediol      
Fusarium culmorum Acetyldeoxynivalenol      
Fusarium equiseti Yavanicin+1      
Fusarium equiseti Acetoxyscirpenediol      
Fusarium equiseti Acetyldeoxynivalenol      
Fusarium equiseti Acetylneosolaniol      
Fusarium graminearum Yavanicin+1      
Fusarium moniliforme Yavanicin+1      
Fusarium moniliforme Acetoxyscirpenediol      
Fusarium moniliforme Acetyldeoxynivalenol      
Fusarium moniliforme Acetylneosolaniol      
Fusarium nivale Acetoxyscirpenediol      
Fusarium nivale Acetyldeoxynivalenol      
Fusarium nivale Yavanicin+1      
Fusarium oxysporum Acetoxyscirpenediol      
Fusarium oxysporum Acetyldeoxynivalenol      
Fusarium oxysporum Acetylneosolaniol      
Fusarium oxysporum Yavanicin+1      
Fusarium
roseum
Yavanicin+1      
Fusarium
roseum
Acetoxyscirpenediol      
Fusarium
roseum
Acetyldeoxynivalenol      
         
         
         
         
         
         
Monographella nivalis Bentenolide      
Penicillium viridictum Ochratoxin      
Penicillium viridicatum Viomellein      
Stachybotrys
Chartarum
Satratoxin F      
Stachybotrys
Chartarum
Satratoxin G      
Stachybotrys
Chartarum
Satratoxin H      
Stachybotrys
Chartarum
Trichothecene   Trichothecenes have multiorgan effects including anoerxia and weight loss, growth retardation, nervous disorders, cardiovascular alterations, immunodepression, hemostatic derangements, skin toxicity, decreased reproductive capacity, bone marrow damage, and alimentary toxic aleukia. After direct dermal application or oral ingestion, the trichothecene mycotoxins can cause rapid irritation to the skin or intestinal mucosa, including skin irritation, burning and itching, rash or blisters, and bleeding. Eye contact can cause tearing, eye pain, conjunctivitis, burning sensations about the eyes, and blurred vision for up to 1 week. Symptoms also include nausea, vomiting, fatigue, dyspnea, and acute vascular effects leading to hypotension and shock.
http://toxsci.oxfordjournals.org/content/104/1/4.full#sec-4

 
 
Stachybotrys
Chartarum
Trichoverrins      
Stachybotrys
Chartarum
Trichoverrols      
Stachybotrys chartarum Verrucarin      
Trichoderma viridi Satratoxin F      
Trichoderma viridi Satratoxin G      
Trichoderma viridi Satratoxin H      
Wallemia sebi walleminol A Unproperly stored fish (too long) on the surface of the fish.  Possible health effects of this mycotoxin are fungal infections http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2268330/  

This information is solely for informational purposes. IT IS NOT INTENDED TO PROVIDE MEDICAL ADVICE. The Authors pf Surviving Toxic Mold take no responsibility for any possible consequences from any treatment, procedure, exercise, dietary modification, action or application of medication which results from reading or following the information contained in this information. The publication of this information does not constitute the practice of medicine, and this information does not replace the advice of your physician or other health care provider. Before undertaking any course of treatment, the reader must seek the advice of their physician or other health care provider.

Is mold making you sick?  Put the effects of Toxic Mold behind you!!

Do you think you might have mold in your home, place of business or school? Are you or someone you love suffering from an unknown illness that doctors can't diagnose?  Is mold making you sick? Go to our Step by Step and start Surviving Toxic Mold.

Get Social With Us

 You can connect with others who have dealt with toxic mold or are still seeking information to help them fight the battle against toxic mold.  Follow us!

We have live discussions taking place everyday on our Facebook group.  Join us on our Facebook group by clicking on the Facebook button below.

Disclaimer

We are not doctors or licensed practitioners.  The advice or suggestions given in the entirety of this website are for informational purposes only.  Please consult your physician before beginning any supplement or regimen. Some statements, treatments or suggestions listed in this website have not been evaluated by the Food and Drug Administration.  This website and/or any of it's contents are not intended to diagnose, treat, cure, or prevent any disease. Read our full disclaimer HERE

Share your Story

Do you have a toxic mold story that you would like to share on SurvivingToxicMold.com?  Send us your story and we will do our best to review it and post it to our "Real Stories" section.  Stories "MUST BE" ready to be published and edited for spelling and grammatical errors.  Please include plenty of photos as well to go along with your story.  Not all stories wil be published.  Stories that are not complete will not be published.

SEND YOUR STORY HERE

Read Jennifer's Story


Read (Founder of Surviving Toxic Mold), Jennifer Cannon's full story of mold exposure, loss & hope for healing and a full recovery HERE

Donate
Has this website helped you?  Are you looking for a way to give back and help support our mission to spread more awareness about the dangers of toxic mold?
READ MORE & DONATE HERE

 

All rights reserved © 2011-2018 Surviving Toxic Mold

Affiliate Disclosure       |      Sources